LUNG CANCER, IT'S EVOLUTION, CAUSES and PRECAUTIONS

LUNG CANCER, IT'S EVOLUTION, CAUSES and PRECAUTIONS


LUNG CANCER, IT'S EVOLUTION, CAUSES and PRECAUTIONS
AUTHORS: Muhammad Mubashir Iqbal, BS Biochemistry GC University Faisalabad, Punjab, Pakistan.

INTRODUCTION

                         
 We know that COPD is linked to lung cancer. After all, the main cause of both diseases is smoking. However, many people do not understand that the link between COPD and lung cancer goes beyond a common cause such as smoking.
COPD is unlikely to produce much more tobacco than COPD than smokers. People with COPD and smoke have a much higher risk of developing lung cancer than those who have the same amount of COPD. Indeed, among smokers, COPD is the biggest factor in developing lung cancer. In addition to COPD being a risk factor for lung cancer,
 there are a number of other important issues (those with COPD and those with lung cancer). COPD may be difficult to recognize the symptoms of lung cancer, and therefore the diagnosis slowed. At the same time, we know that lung cancer prognosis is better and earlier it is diagnosed. When lung cancer is diagnosed, COPD may interfere with the possible treatments for cancer. What do you need to find out if you have COPD, lung cancer, or both?

WHAT IS COPD?

COPD is a disease group characterized by the obstruction of airways in the lungs. In contrast to the recoverable air road (as with asthma) the obstruction of COPD is not fully portable to treatment.
Although the most commonly used COPD term to describe a mixture of emphysema and chronic bronchitis, diseases are classified as COPD.
COPD-INDEPENDENT LUNG CANCER RISK FACTOR
COPD is not only a risk factor for lung cancer, but is perhaps the biggest risk factor. With being an "independent" risk factor it is possible that something could increase lung cancer alone - all by itself. COPD would have a high chance of developing lung cancer than the person without COPD.


There are more than a dozen studies that have found that COPD is an independent risk factor for lung cancer, although the amount of risk changes slightly and rises with an increased risk of 2 times a 10-hour increased risk. Overall, COPD appears to increase the risk of COPD 2 times to 4 times in view that smokers must be moderate and never smokers, and even more in smoking.

STATISTICS AND FREQUENCY

Looking at statistics about COPD and lung cancer, it is important to note the first time these diseases occur in the United States.

Cancer is the cause of deaths in the United States (after heart disease) and lung cancer is the only cause of cancer-related deaths in both men and women. COPD is now thought to be the third or 4th cause of death in the United States. About 11 million Americans were diagnosed with COPD and an increasing number of Americans are living with undiagnosed COPD. This translates to eight to ten percent of the population having a certain degree of COPD rising to ten to twenty percent of smokers.

DISEASE PROGRESS


COPD will increase the risk of lung cancer regardless of smoking, but how does this happen? There are a number of theories.
One theory is that genetic factors are common for both COPD and lung cancer. In other words, genetic acceptability of overlapping makes it more likely that some people may develop these diseases.
Another theory is that the airways would cause cancer-causing substances (carcinogens) to destroy the cilia in the airways that resulted in COPD. The cilia are small structures like hair in the airways that contribute to removing toxins from their way into the airways. Use these cilia wavelike movement to release tiny particles up and out of the airways in which they can be swallowed. Cigarette cigarettes contain some chemicals and destroy the cilia. By continuing in the airways, these carcinogens can cause the changes that lead to the emergence of a healthy lung cell as a cancer cell..
Another theory is that lung cancer may be the result of chronic inflammation in the airways associated with COPD. Chronic cancer can sometimes lead to cancer, such as with esophageal cancer and cervical cancer.

EMPHYSEMA: 


This is a chronic lung disease, characterized by damage to alveoli, the tiny sacs at the end of the airways that exchange oxygen and carbon dioxide.
Chronic bronchitis -
Chronic fertaitite is a condition through inflammation and scaling of the airways. This inflammation has resulted in mucus collecting in the airways, overcrowding the airways and often recurring infections.

SYMPTOMS:


Lung cancer in the early stages of survival can increase significantly, but until we have an extensive screening test for everyone, we often have to rely on recognizing early signs and signs of lung cancer.

The individuals with COPD show the normal symptoms as symptoms of lung cancer which is a serious issue i.e. both diseases cause persistent cough, short breath, pulmonary infections again and even weight loss.

The more common type of lung cancer today can make this distinction more difficult. In the past, squamous cell carcinoma of the lungs and small cell lung cancer were more common. These cancers often grow close to the large lungs of the lungs and cause symptoms such as blood turns (less common with COPD).

In contrast, lung adenocarcinoma is the most common type of lung cancer diagnosed today. These cancers often grow on the edge of the lungs, and therefore breathing is often increased, the first donation is often an activity. This dyspnea (short breath sensation) found with COPD can be very similar to the shortest breath that may cause lung cancer.

TREATMENT

COPD is not only a risk factor for COPD (and the diagnosis is made more difficult) but the treatment of lung cancer can be more difficult. People with COPD may not be able to tolerate treatments such as lung cancer surgery or radiation therapy that may have lung volume.

Keep in mind that treatment and even surgery exists, however, for people with mild or moderate COPD. If you have COPD and lung cancer is diagnosed, make sure you get a doctor who will work with your COPD to treat your lung cancer.



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